Clinical Neurophysiology is the official journal of the International Federation of Clinical Neurophysiology, the Brazilian Society of Clinical Neurophysiology, the Czech Society of Clinical Neurophysiology, the Italian Clinical Neurophysiology Society and the International Society of Intraoperative Neurophysiology.The journal is dedicated to fostering research and disseminating information on all aspects of both normal and abnormal functioning of the nervous system. The key aim of the publication is to disseminate scholarly reports on the pathophysiology underlying diseases of the central and peripheral nervous system of human patients. Clinical trials that use neurophysiological measures to document change are encouraged, as are manuscripts reporting data on integrated neuroimaging of central nervous function including, but not limited to, functional MRI, MEG, EEG, PET and other neuroimaging modalities.
The Journal has special emphases on epilepsy and on studies of cognitive function and cognitive disorders. Motor neurone and neuromuscular diseases, vestibular disorders, motor control and somatosensory physiopathology are also covered by the Journal. Studies on animals and technical notes must have clear relevance and applicability to human disease, and studies reporting normative data for specific tests must have clear novelty. Case Reports are not generally accepted as full length submissions but may be considered as peer-reviewed Letters.
|From Kirkpatrick's Clinical Practice Guidelines|
Deficient muscle activation in patients with Complex Regional Pain Syndrome and abnormal hand postures: An electromyographic evaluation.
- [PubMed - as supplied by publisher]Other articles that may elucidate the matter further:Stretch reflex responses in Complex Regional Pain Syndrome-related dystonia are not characterized
Antagonist motor responses correlate with kinesthetic illusions induced by tendon vibration.
Thalamic single neuron activity in patients with dystonia: dystonia-related activity and somatic
[A case of post-hemiplegic painful dystonia following thalamic infarction with good response to botulinus toxin].
Functional anatomy of thalamus and basal ganglia.